Anti-Wilms Tumor Protein antibody
| Name | Anti-Wilms Tumor Protein antibody |
|---|---|
| Supplier | Abcam |
| Catalog | ab15249 |
| Prices | $382.00 |
| Sizes | 500 µl |
| Host | Rabbit |
| Clonality | Polyclonal |
| Isotype | IgG |
| Applications | IP IHC-P |
| Species Reactivities | Mouse, Human, Rat |
| Antigen | Synthetic peptide (Human) (C terminal) |
| Description | Rabbit Polyclonal |
| Gene | WT1 |
| Conjugate | Unconjugated |
| Supplier Page | Shop |
Product images
Immunohistochemical analysis of murine kidney tissue, staining Wilms Tumor Protein (red) with ab15249 at 1/300 dilution. A fluorescent-conjugated secondary antibody was used to detect staining.
Product References
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TAp73 is essential for germ cell adhesion and maturation in testis. - TAp73 is essential for germ cell adhesion and maturation in testis.
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aPKClambda/iota and aPKCzeta contribute to podocyte differentiation and - aPKClambda/iota and aPKCzeta contribute to podocyte differentiation and
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Vps34 deficiency reveals the importance of endocytosis for podocyte homeostasis. - Vps34 deficiency reveals the importance of endocytosis for podocyte homeostasis.
Bechtel W, Helmstadter M, Balica J, Hartleben B, Kiefer B, Hrnjic F, Schell C, Kretz O, Liu S, Geist F, Kerjaschki D, Walz G, Huber TB. J Am Soc Nephrol. 2013 Apr;24(5):727-43.
Recovery of urinary nanovesicles from ultracentrifugation supernatants. - Recovery of urinary nanovesicles from ultracentrifugation supernatants.
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Role of the polarity protein Scribble for podocyte differentiation and - Role of the polarity protein Scribble for podocyte differentiation and
Hartleben B, Widmeier E, Wanner N, Schmidts M, Kim ST, Schneider L, Mayer B, Kerjaschki D, Miner JH, Walz G, Huber TB. PLoS One. 2012;7(5):e36705.
Autophagy influences glomerular disease susceptibility and maintains podocyte - Autophagy influences glomerular disease susceptibility and maintains podocyte
Hartleben B, Godel M, Meyer-Schwesinger C, Liu S, Ulrich T, Kobler S, Wiech T, Grahammer F, Arnold SJ, Lindenmeyer MT, Cohen CD, Pavenstadt H, Kerjaschki D, Mizushima N, Shaw AS, Walz G, Huber TB. J Clin Invest. 2010 Apr;120(4):1084-96.
Loss of podocyte aPKClambda/iota causes polarity defects and nephrotic syndrome. - Loss of podocyte aPKClambda/iota causes polarity defects and nephrotic syndrome.
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